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The mechanism of ATRA’s inductive effect on GS expression required ERK1/2 signaling but the mechanism is not clear, as ATRA treatment did not influence the expression of Nrf2 or NFκB and it had no effect on the GS promoter activity [141]

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GHK-Cu supports this transition at the tissue level by modulating the gene expression patterns associated with the metabolically inflamed state that obesity research consistently identifies in adipose tissue