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Iron ions in the form of Fe 2+ or Fe 3+ reside in cells i n vivo, and iron homeostasis maintains the relatively stable content of these ions through transferrin receptor 1 (TFR1)-regulated iron transport, ferroportin 1 (FPN1)-mediated iron output storage, and ferritin-regulated iron storage (Fig
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Vit-E could effectively prevent ferroptosis by reducing the Fe 3+ center to inactive Fe 2+ in lipoxygenase (LOX), thereby inhibit enzyme activity of LOX
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