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Biologists suspect that when the epigenome accumulates too many marks, over time, the signals it sends to cells change dramatically—and that those new signals produce the effects of aging
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Collectively, this study identifies GGCT as a metabolic switch that diverts Gln flux toward GSH synthesis to maintain redox homeostasis, while enhancing glucose-fueled anaplerosis into the TCA cycle to sustain cell proliferation
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