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At the cellular level, GHK influences key processes that determine myofibroblast behavior during repair: Limits myofibroblast persistence: GHK is associated with pathways that favor myofibroblast apoptosis or deactivation during later repair phases
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Introducing randomized residues on this type of sequence enables the construction of CKP-derived cyclic peptides, which can then be screened by phage display
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