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There are results indicated that the Nrf2 gene has an NF-κB binding site which p65/p50 heterodimer is recruited, and IκK-β plays a core role in NF-κB signaling, contains an ETGE motif that enables it to bind to Keap1 (Lee et al., 2009

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It’s slated to last through 2025 and is still is listed as still recruiting patients